Trigger finger, medically known as stenosing flexor tenosynovitis, is a common musculoskeletal condition affecting the flexor tendons of the fingers and potentially compromising hand mobility and functional performance. It is characterized by impaired tendon gliding during finger movement, resulting in pain, clicking, catching, or locking during flexion or extension. Symptoms may initially be mild and intermittent but can progressively interfere with daily activities, including gripping, writing, and handling objects. The condition most commonly involves thickening of the first annular pulley (A1 pulley), together with structural changes in the flexor tendon or surrounding tissues, leading to restricted tendon excursion and impaired gliding. Several predisposing factors have been associated with trigger finger, including diabetes mellitus, advancing age, and certain joint and connective tissue disorders. Repetitive forceful gripping may aggravate symptoms, although it cannot be considered a definitive cause in every case. The condition may affect one or multiple digits and may occur in one or both hands, including the thumb. Common clinical manifestations include localized pain and tenderness at the base of the affected finger, clicking during movement, and a sensation of catching or locking, particularly during flexion or extension. Symptoms may be more noticeable in the morning or following periods of inactivity. In advanced cases, patients may need to use the opposite hand to extend the affected digit, and persistent symptoms may result in joint stiffness, restricted range of motion, reduced grip efficiency, and functional limitations. Diagnosis is primarily clinical and is based on the patient's history and physical examination, including assessment of pain, range of motion, palpation of the A1 pulley region, and observation of tendon movement during active finger flexion and extension. Ultrasonography may be useful when the diagnosis is uncertain or further assessment of the tendon and pulley is required. Differential diagnoses include Dupuytren's disease, inflammatory joint disorders, and traumatic conditions affecting finger movement. Treatment depends on symptom severity, duration, and functional impairment. Conservative management may include activity modification, avoidance of forceful repetitive gripping, and appropriate splinting when indicated. Analgesic or nonsteroidal anti-inflammatory medications may help relieve pain when medically appropriate, although they do not necessarily correct the underlying mechanical restriction. Local corticosteroid injection is a commonly used treatment option and may provide substantial symptom relief; however, treatment response varies among individuals, and recurrence may occur. Particular caution is required in patients with diabetes because corticosteroid injections may cause temporary elevations in blood glucose levels. Persistent, severe, or functionally limiting cases, particularly those associated with significant locking, may require surgical release of the A1 pulley to restore tendon gliding. Physiotherapy plays an important role in the management of trigger finger by helping reduce symptoms, improve mobility, and restore hand function. Rehabilitation may include patient education, activity modification, appropriate splinting, gentle range-of-motion exercises, and tendon-gliding exercises tailored to the patient's clinical presentation and tolerance. Exercises should be performed carefully, avoiding forceful movements or repetitive attempts to overcome painful catching or locking, as these may aggravate tissue irritation. Aggressive massage and repeated direct pressure over the affected pulley should also be avoided. Following surgical intervention, rehabilitation focuses on pain and swelling management, preservation of finger mobility, restoration of tendon gliding, and gradual recovery of strength and functional hand use, according to the surgeon's instructions and wound status. Although most patients achieve favorable outcomes with appropriate management, prolonged symptoms may contribute to persistent pain, joint stiffness, and reduced hand function. Potential treatment-related complications include persistent or recurrent symptoms, postoperative scar tenderness, stiffness, infection, and, rarely, injury to adjacent structures. Therefore, appropriate medical follow-up and individualized rehabilitation are essential for optimizing outcomes and minimizing complications. In conclusion, trigger finger is a mechanical and structural disorder of the digital flexor tendon system, most commonly associated with thickening of the A1 pulley and impaired tendon gliding. Early diagnosis and appropriate treatment selection are important for achieving favorable clinical outcomes. Physiotherapy and functional rehabilitation contribute to pain reduction, improved mobility, and restoration of hand performance, both during conservative management and following surgery. An individualized, evidence-informed approach integrating medical treatment and rehabilitation remains essential for effective patient care.